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| 1.
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Nakayama, Kenji ; Takahashi, Yutaka ; Sato, Yayoi ; Nukada, Yasuaki
概要:
金沢大学大学院自然科学研究科情報システム<br />金沢大学工学部<br />An adaptive switched-capacitor (SC) line equalizer system that can be applied to
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four-wire full-duplex and multirate digital transmission is described. Several kinds of noises can exist in programmable high-gain SC equalizers, such as switching noise, DC offset jump, and transient response. To avoid their effects, an adaptive SC filter is proposed. Two identical SC circuits are used in parallel. An input signal is fed into both SC circuits, and output signals from both circuits are alternately sent after the above noises are eliminated. Furthermore, many different data rates can be handled by slightly modifying an equalizer circuit. A bridged-tap echo canceller and a DC offset canceller are modified so as to be applied to the proposed adaptive SC filter. A line equalizer system, that handles data rates ranging from 3.2 to 64 kb/s was designed. An LSI was fabricated using a 3-μm CMOS process. Experimental results show that noise effects are mostly eliminated, and frequency responses and eye openings are close to the designed values.
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Nakayama, Kenji ; Takahashi, Yutaka ; Satoh, Yayoi ; Naka, Masahiro ; Nukada, Yasuaki
概要:
The authors present a single-chip switched capacitor (SC) line equalizer system which can be applied to full duplex digital transmission. Adaptive SC filters, constructed with programmable capacitor arrays (PCAs), usually cause undesired
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responses, such as spike noise and transient response, which degrade data transmission quality. To avoid these phenomena, a duplex SC equalizer which has the same circuits in parallel is introduced. The PCAs in one duplex equalizer, whose output is not transferred, are varied. After the undesired phenomena vanish, the output is alternated. The equalizer system can be applied at several different bit rates by changing the external control signals. Algorithms and circuit realizations for a bridged tap echo canceller and a dc offset canceller are further improved.
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Kuranishi, Yoshiaki ; Takahashi, Yutaka ; Nakayama, Kenji ; Senba, Takashi
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金沢大学理工研究域 電子情報学系
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Shakoori, Abbas ; Mai, Wei ; Miyashita, Katsuyoshi ; Yasumoto, Kazuo ; Takahashi, Yutaka ; Ooi, Akishi ; Kawakami, Kazuyuki ; Minamoto, Toshinari
概要:
The authors' recent discovery that glycogen synthase kinase-3β (GSK-3β) participates in colon cancer cells' survival and
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proliferation prompted us to investigate whether GSK-3β inhibition alters proliferation of colon cancer cells in vivo. Groups of four or five athymic mice (Balb/c, nu/nu) with subcutaneous xenografts of SW480 human colon cancer cells were treated with dimethyl sulfoxide (DMSO) or different doses (1, 2 and 5mg/kg body weight) of either small-molecule GSK-3β inhibitor (SB-216763 and AR-A014418) by intraperitoneal injection three times per week for 5 weeks. Compared with DMSO (a diluent of the GSK-3β inhibitors) as a control, either GSK-3β inhibitor significantly inhibited proliferation of cancer cell xenografts in the rodents in a dose-dependent manner. Histochemical and immunohistochemical analysis of tumor xenografts demonstrated a significant, dose-dependent decrease in fractions of proliferating cells and an increase in the incidence of apoptosis of cancer cells in mice treated with either GSK-3β inhibitor. No adverse events or effects were observed in the rodents during the course of treatment, except for rare lethal accidents due to intraperitoneal injection. Morphological examination showed no apparent pathologic changes in major organs including the lungs, liver, pancreas, kidneys, spleen and large bowel of rodents treated with DMSO and the GSK-3β inhibitors. The results indicate that the GSK-3β inhibitors would be a novel class of therapeutic agent for colon cancer. © 2007 Japanese Cancer Association.
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Takahashi, Yutaka ; Yamashita, Kaname
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Takahashi, Yutaka ; Yasumoto, Kazuo
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Omote, K. ; Kitahata, H. ; Yamashita, Kaname ; Yasumoto, Kazuo ; Takahashi, Yutaka
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Yasumoto, Kazuo ; Koizumi, K. ; Saiki, I. ; Takahashi, Yutaka
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Yamashita, Kaname ; Kitahara, Hidekazu ; Yasumoto, Kazuo ; Omote, Kazuhiko ; Takahashi, Yutaka ; Minamoto, Toshinari
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Kitakata, H. ; Takahashi, Yutaka
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